Similarly, exogenous SLC9A6‐126aa markedly exacerbated HFD‐induced obesity, elevated the fasting plasma glucose levels, and resulted in abnormal serum biochemical indices similar to those observed with exogenous circ‐SLC9A6, whereas no significant change was observed in the AAV9‐TBG‐mut‐circ‐SLC9A6 group (Figure 4E–H). This evidence concerns the gene SLC9A6 and obesity disorder.