In details, oxidized CaMKII contributes to apoptosis post-myocardial infarction and atrial fibrillation,246 while O-GlcNAcylation contributes to hyperglycemia-induced arrhythmia.253–256 Whereas, nitrosylation of CaMKII confers a sex-dependent protective effect against harm from ischemia/reperfusion in females.257. The gene discussed is CAMK2G; the disease is myocardial infarction.