NGF and myocardial infarction: M2 macrophages suppress ventricular arrhythmia caused by sympathetic nerve remodeling after myocardial infarction by reducing the expression of NGF (Liu et al., 2023; Yang et al., 2024) found that Sinapic acid (SA) stimulated the PPARγ pathway with concentration-dependent to promote M2 polarization and reduce NGF (Yang et al., 2022).