Interestingly, 3‐TYP treatment (the AβO + HKL + 3‐TYP group) attenuated the viability of the hippocampal neuronal model of AD, when compared with the AβO + HKL group (p < 0.05) (Figure 7A: F = 85.68, df = 3 for 24 h; F = 98.39, df = 3 for 48 h). The gene discussed is ABO; the disease is Alzheimer disease.