Results showed that Cre+CPT1Afl/fl mice receiving LPS + rIL‐10 administration had less airway inflammatory cells, especially macrophages and neutrophils aggregation, as well as decreased IFN‐γ, IL‐6, G‐CSF, CXCL1/KC levels in Balf and serum, compared with Cre+CPT1Afl/fl ALI mice without IL‐10 supplement (Figure 6a–c). The gene discussed is IFNG; the disease is acute respiratory distress syndrome.