Myocardial infarction is characterized by an increase in type 17 helper T cells (Th17) and a decrease in Tregs, resulting in the overproduction of pro‐inflammatory cytokines such as IL‐17A, IL‐17F, and IL‐22,179 and these cytokines are positively correlated with serum creatine kinase isoenzyme (CK‐MB), which is a recognized marker of myocardial infarction.180. The gene discussed is IL17A; the disease is myocardial infarction.