TNFRSF14 and glioblastoma: Furthermore, transfection of FAK wild type overexpression vector in GBM cells obviously restored their reduced phosphate-FAK (p-FAK) and phosphate-p65 (p-p65) expression induced by TNFRSF14 knockdown, while FAK Y397F mutant vector couldn’t exert this role (Fig. 4F, G and Fig S4F).