rhCXCL1/CXCL5 treatment induced elevated expression of CD163 and CD206, and reduced CD80, CD86 and IL-6 expression in THP-1 derived macrophages relative to the incubation of CM from TNFRSF14-knockdown GBM cells (Fig. 5F). This evidence concerns the gene CXCL5 and glioblastoma.