STAT3 and hepatocellular carcinoma: The expressions of matrix metalloproteinase-2, matrix metalloproteinase-9, p-STAT3, STAT3, Snail, β-catenin, and c-Myc decreased after the treatment with brevilin A, indicating the inactivation of STAT3/Snail and Wnt/β-catenin signaling pathways was involved in the anti-HCC activity of brevilin A.