Previous studies have used ABEmax to insert an A•T to G•C change in the CREs of BCL11A, intergenic HBS1‐like translational GTPase (HBS1L) to MYB proto‐oncogene, transcription factor (TF) (MYB) region, KLF1, and β‐like globin genes controlling erythrocyte HbF expression to quantify their impact on γ‐globin gene regulated HbF expression, increases in HbF levels, and provide potential therapeutic insights for SCD.246. The gene discussed is MYB; the disease is Schnyder corneal dystrophy.