Additionally, LGE detects marked expansion of the extracellular space associatedwith amyloidosis (amyloid deposition and fibrosis) and the LGE pattern canpotentially differentiate Amyloid-Transthyretin (ATTR) from AL patients [28], but there are no studiesregarding the role of LGE in arrhythmogenesis in these diseases. The gene discussed is TTR; the disease is amyloidosis.