Among the inflammatory CC chemokines regardingHF, C-C motif chemokine ligand 4/major capsid protein-1 (CCL2/MCP-1) has the ability to inhibit cardiac remodeling and preventpersistent myocardial damage leading to ischemic necrosis, and has been suggestedin numerous studies as a potential therapeutic target for HF [31, 32]. The gene discussed is CCL2; the disease is hydrops fetalis.