IL33 and congenital rubella syndrome: Soluble suppression of tumorigenicity 2(sST2), a member of the IL-1 receptor family, is released by cardiomyocytes andpulmonary endothelial cells and contributes distinctively to organ fibrosis [21].Not only does sST2 present potential in predicting AKI and CRS in acute cardiacevents, but IL-33/ST2 axis induces fibrosis which is appealing as an emergingmechanistic bio profile in CRS [21].