SARS-CoV-2 employs human angiotensin-convertingenzyme 2 (ACE2) as cell entry receptor, further infecting lung, heart, liver, andother organs, and leading to blood coagulopathy and organ dysfunction [7, 8, 42].Nevertheless, no connection has been reported so far between the usage ofanti-hypertensive medications and increased COVID-19 susceptibility, severity, ormortality [43, 44]. This evidence concerns the gene ACE2 and COVID-19.