In the stage of myocardial ischemia, insufficient exogenous energysupply can cause cardiomyocytes to supply amino acids and fatty acids to maintaincell survival through adenosine monophosphate-activated protein kinase (AMPK)mediated mammalian target of rapamycin (mTOR) dependent autophagy of rapamycin,which has A cell protection effect [11]. This evidence concerns the gene MTOR and myocardial ischemia.