SLC7A11 and atrial fibrillation: Liu et al. [38] additionally noted thatexosomes derived from fibroblasts during cardiac pacing were capable of promotingferroptotic induction in atrial fibrillating cardiomyocytes via exo-miR-23a-3psecretion, which is, in turn, able to target SLC7A11 and the Xc- system.This additionally suggests that efforts to inhibit the release of exosomes mayhelp protect against susceptibility to AF [39].