Myricetin possessespotential protective effects in DCM, which attributed to alleviate oxidativestress, inflammation, apoptosis, and fibrosis.The underlying mechanisms of it atleast partly associated to the inhibition of theIκB-α/NF-κB/p65 and TGF-β/Smad signalingpathways and enhancing the expression of Nrf-2 [80]. The gene discussed is NFE2L2; the disease is familial dilated cardiomyopathy.