AKT1 and myocardial infarction: HIF-1α/miR-126-3p axis was involved inET-induced myocardial angiogenesis post-MI by regulating PI3K/protein kinase B (AKT)/endothelial nitric oxide synthase (eNOS) and mitogen-activated protein kinase (MAPK)signaling pathways, thereby improving cardiac function against myocardial injury[68].