Mechanisms for developing myocardial injury in thesetting of COVID-19 infection are not fully understood, but one hypothesis isvirus-mediated lysis of cardiomyocytes, which has also been observed in otherviral infections, or as a consequence of SARS-CoV-2 binding angiotensinconverting enzyme 2 (ACE2) receptors in the heart [39]. Here, ACE2 is linked to infection.