AR and polycystic ovary syndrome: Indeed, the induction of a classic PCOS‐like reproductive and metabolic phenotype in F0 dams was successfully counteracted by blocking the androgen receptor pathway, in line with previous research.[17, 18] Moreover, in utero hyperandrogenism resulted in a global reduction of the transcriptional activity in the placentas leading to diminished placental labyrinth formation as well as impaired proliferation and differentiation of trophoblast precursors.