ETS1 and nonpapillary renal cell carcinoma: Since the above results indicated that acetylation of ETS1 at the K245 site was important for modulating the sensitivity of ccRCC to sunitinib (Figures 4 and 5), we were curious about whether there are other posttranslational modifications around the ETS1‐K245 site that are involved in modulating the acetylation of ETS1‐K245 or the sensitivity of ccRCC to sunitinib.