Myelin oligodendrocyte glycoprotein (MOG) antibody‐associated disease (MOGAD) is a demyelinating disorder that affects children and adults, and may be associated with recurrent or bilateral optic neuritis, acute disseminated encephalomyelitis, transverse myelitis, brainstem, cortical, or leptomeningeal involvement.1, 2, 3 Recently proposed diagnostic criteria highlight the importance of an appropriate clinical association, MOG antibody seropositivity by cell‐based assay, and exclusion of an alternate etiology.3 This evidence concerns the gene MOG and transverse myelitis.