It seems that anemia is the result of a combination of factors [2], such as the involvement of the bone marrow in which iron is abnormally reused [3], abnormal metabolism of iron in malignant and chronic inflammatory diseases [4], the inadequate response of the bone marrow to the action of erythropoietin [5,6], and the inhibition of erythropoiesis by certain inflammatory mediators such as TNF, IL-1, IL-10 [7-9], IL-6 [7,10] and interferon γ [11]. This evidence concerns the gene IL10 and anemia (phenotype).