Yaouzu et al. have explored the molecular mechanisms of BA antiproliferative effects against U87MG and A172 glioblastoma cells revealing the downregulation of the NF-κB pathway and upregulation of caspase-3 and -9, thus suggesting that apoptosis occurred through mitochondria-mediated mechanisms [59]. The gene discussed is NFKB1; the disease is glioblastoma.