In order to further explore other potential mechanisms behind GFP in skeletal muscle atrophy of T2DM, we predicted the possibility of the interaction of five monosaccharides of GFP with TNF-α, NF-κB, FBXO32, and TRIM63 protein targets via molecular docking with MOE2019 software. This evidence concerns the gene FBXO32 and type 2 diabetes mellitus.