Oxytocin receptor knockdown reduces the viability of glioma cells [115], neurotensin 1 receptor knockdown suppresses the growth and invasion of these cells [54], and the silencing of the truncated splicing variant of the somatostatin 5 receptor sensitizes glioblastoma cells to the antitumor action exerted by the somatostatin analog pasireotide [103]. Here, SSTR5 is linked to glioblastoma.