IGLON5 and glycogen storage disease VI: Finally, a notable mention is made of several patients’ case reports with clinical and serological evidence of IgLON5-IgG disease without degenerative features like hyperphosphorylated tau in brain tissue, which may shed light on the sequence of pathogenesis of this disease entity, specifically, that tau accumulation occurs later in the disease course and is a consequence of antibody-mediated neuronal dysfunction [15,40,41,42,43].