Therefore, investigating the fundamental molecular processes of myogenesis, involving potential regulatory genes such as Mustn1, is significant for devising future comprehensive strategies to prevent and treat skeletal muscle wasting (e.g., diabetes, sarcopenia, dystrophies) and injuries arising from direct (e.g., lacerations, contusions, and strains) and indirect (e.g., ischemia and neurological dysfunction) causes [87,88]. This evidence concerns the gene MUSTN1 and sarcopenia.