Specifically, ECs import miR-376c into NSCLC cells via EVs, which then target intracellular YTHDF1 and inhibit its expression, thereby disrupting the Wnt/β-catenin pathway mediated by YTHDF1 and ultimately leading to the inhibition of cell proliferation [147]. The gene discussed is YTHDF1; the disease is non-small cell lung carcinoma.