They found that hsa-mir-30b-5p was downregulated in B cells in pSS patients compared with control samples, and hsa-mir-30b-5p expression in B cells was inversely correlated with B cell-activating factor (BAFF), which harbors an hsa-miR-30b-5p binding site in the 3′UTR. The gene discussed is TNFSF13B; the disease is peeling skin syndrome.