Because N-glycans of B7-H3 from Ca9-22 oral cancer cells have been shown to have higher amounts of fucosylation than normal cells [61], Huang et al. identified that direct core fucosylation by FUT8 maintains the stability of B7-H3 in TNBC cells and mediates immunosuppression, which is relevant for patients with TNBC with a poor prognosis [60,62]. This evidence concerns the gene CD276 and lip and oral cavity carcinoma.