CRP and atrial fibrillation: The chronic inflammation in patients with heart failure, characterised by elevated levels of pro-inflammatory molecules such as interleukin-6 (IL-6), tumour necrosis factor-alpha (TNF-α), and C-reactive protein (CRP), triggers atrial fibrosis, enlargement, and cell death, disrupting the normal organisation and function of atrial tissue and leading to the development and maintenance of AF.