AKT1 and endometriosis: KEGG enrichment analysis revealed that nitrogen metabolism [72], PI3K-Akt [73], platelet activation [74], the NOD-like receptor signalling pathway [75], ECM-receptor interactions [76], cytokine‒cytokine receptor interactions [76], IL-17 signalling [77], complement and coagulation cascades [78], TNF signalling and proteoglycans in cancer [79] have been implicated in the pathogenesis of endometriosis.