NFKB1 and colitis: We reasoned that the molecular redundancies between RelB NF-κB factors obscured the phenotypic penetrance of Nfkb2∆CD11c mice in the acute colitis regime and that repeated DSS exposure unmasked colitis-resilient phenotypes in these mice because of exaggerated DC-mediated immune reactions in the chronic regime (Wirtz et al, 2017).