Martayan et al. [47] assessed the role of B2m in regulating the conformation and surface expression of HLA-C molecules on a B2m-deficient kidney carcinoma cell line with the three mAbs L31, W6/32, and Q/128, reactive to alpha 1, 2, and 3 domains of the HCs, respectively. Here, B2M is linked to renal carcinoma.