Kollnberger’s team [144] further compared the affinity of KIR and LILR to B2m-associated and B2m-free HC monomers and dimers of HLA-B*27:05, which is associated with ankylosing spondylitis (AS), and HLA-B*27:09, which is not associated with AS. This evidence concerns the gene HLA-B and ankylosing spondylitis.