The mechanisms involved include the modulation of cell cycle regulatory proteins [46,47], the inhibition of angiogenesis [48,49], and the modulation of key signaling pathways, such as the PI3K/AKT/mTOR [50] and PI3K/Akt/FoxO 3a pathways [35,51], which are crucial for cancer cell survival and proliferation. The gene discussed is AKT1; the disease is cancer.