It was also by inhibiting a cell death mechanism that Vitexin demonstrated potential in the treatment of rats with diabetic nephropathy, by attenuating renal fibrosis, oxidative damage, and ferroptosis through the activation of GPX4 (glutathione peroxidase 4) (Zhang et al., 2023), revealing yet another indicator of Elo's multi‐action pathway. The gene discussed is GPX4; the disease is renal fibrosis.