In studies related to ovarian cancer cells, gallic acid demonstrated its ability to arrest cell cycle progression at S/G2 phase by reducing levels of cell cycle-related proteins CDC2, p-Cdc2, and cyclin B. Additionally, it activated an intrinsic apoptotic pathway mediated by Caspase-3 through upregulation of p53 (44). Here, CDK1 is linked to ovarian carcinoma.