TRPM2 and non-small cell lung carcinoma: Enforced expression of ectopic TRPM2 in sensitive EGFRm NSCLC cells abrogated the ability of osimertinib to induce ROS generation, DNA damage, and apoptosis, whereas genetic inhibition of TRPM2 using gene knockdown in osimertinib‐resistant cell lines restored the effect of osimertinib on induction of ROS generation, DNA damage, and apoptosis.