These pathways included increased activity of inflammation-related pathways (interferon alpha response, interferon gamma response, mTORC1 signaling, and TNFα signaling via NFκB), steroid response-related pathways (cholesterol homeostasis and androgen and estrogen response), and cancer progression-related pathways (epithelial mesenchymal transition, apical junction, and adipogenesis). The gene discussed is IFNG; the disease is cancer.