Hepcidin expression decreases in several diseases associated with iron overload such as inefficient erythropoiesis (e.g., thalassemia) or hereditary hemochromatosis (HH), whereas excessive hepcidin can lead to anemia of inflammation or iron‐refractory iron deficiency anemia (IRIDA).78 The gene discussed is HAMP; the disease is hereditary hemochromatosis.