ATP11A and idiopathic pulmonary fibrosis: We subsequently found evidence of colocalization between the disease GWAS and the ATP11A-eQTL signals (PP4 = 0.996 with IPF GWAS; 0.995 with COVID-19 GWAS; Figure 4B–C; Table S9) but lack of colocalization between the ATP11A-eQTL signals from the Lung eQTL Study and GTEx lung (PP4 = 0.344; Table S9).