While the evidence we have presented suggests that variants in ATP11A and DPP9 impact COVID-19 and IPF pathogenesis by altering expression of these genes, recent evidence from Nakanishi et al. demonstrated that the sQTL data for these loci also colocalize with severe COVID-19.101 Future studies will be necessary to understand the effect of these risk variants on splicing, but we note that each sQTL is predicted to only impact a single non-canonical isoform based on Ensembl annotation. This evidence concerns the gene ATP11A and idiopathic pulmonary fibrosis.