Increased levels of proinflammatory cytokines, such as interleukin (IL)‐18, have been shown in patients suffering from chronic lung diseases (Chung, 2001; Imaoka et al., 2008), who are prone to develop both alveolar hypoxia and pulmonary hypertension (Gu et al., 2023). This evidence concerns the gene IL18 and pulmonary arterial hypertension.