TGFB1 and COVID-19: The post‐mortem histopathological analyses have suggested persisting fibrotic abnormalities; however, as discussed by Ntatsoulis et al., large‐scale, long‐term follow‐up studies are necessary to clarify the link between COVID‐19 and interstitial lung alterations because, on the one hand, SARS‐CoV‐2 infection induces pro‐fibrotic factors such as the transforming growth factor‐β1 (TGFβ1).