Bone marrow transplantation or bone marrow-specific overexpression of MYDGF attenuated leukocyte homing in the aortas of atherosclerotic mice and ameliorated atherosclerosis by reducing inflammation and endothelial cell damage via the PKCδ/MAP4K4/NF-κB pathway (Meng et al. 2021). The gene discussed is MYDGF; the disease is atherosclerosis.