By using CD69 and CD103 as markers of NK cell tissue-residency (Hervier et al, 2019), we found that both circulating (CD56+CD69– or CD56+CD103–) and lung-resident (CD56+CD69+ or CD56+CD103+) NK cells showed upregulated LILRB1 expression within granulomatous lesions, and we also noticed that these NK cells displayed relatively higher LILRB1 expression in necrotic granulomas than that in non-necrotic granulomas (Fig. 3I–K). This evidence concerns the gene NCAM1 and Granuloma.