According to research, in mice and humans with ALI, the recruitment of CD4 + CD25 + FOXP3+ Treg cells in the alveoli, particularly through the leukotriene B4(LTB4)‐leukotriene B4 receptor (BLT1) pathway,199 could decrease lung fibroproliferation200 and improve lung inflammation.201. Here, FOXP3 is linked to acute respiratory distress syndrome.