However, in R‐MCF7/R‐ZR75.1 cells, combining targeted inhibition of AKT1 (MK2206 or siAKT1) and activators of STING signaling (ADU‐S100 or HT‐DNA) significantly increased the percentage of DCs that engulfed CFSE‐labeled tumor cells much more than activators of STING signaling monotherapy (Figure 7G,H; and Figure S7E,F, Supporting Information). This evidence concerns the gene STING1 and neoplasm.