NFKB1 and infection: Furthermore, we performed chromatin immunoprecipitation (ChIP) assay and found that, compared to no infection, ΔcagA infection, or pcDNA3.1 transfection, WT H. pylori infection and cagA-pcDNA3.1 transfection significantly increased NF-κB binding to the ANGPTL4 promoter in AGS cells, which were abolished when these cells were pretreated with BAY 11-7082 (Fig. 2M and Fig. S2I).