Our data indicated that he administration of STZ-induced T1DM mice with sodium citrate buffer, insulin and adoptively transferred dBMCs significantly decreased the level of Zinc transporter 8 antigen protein (ZenT8) comparing to that in naive mice received sodium citrate buffer (0.47 ± 0.03 pg/mg, 0.97 ± 0.08 pg/mg and 1.36 ± 0.03 pg/mg, respectively versus 2.37 ± 0.14 ng/mg) (Fig. 5C). This evidence concerns the gene INS and type 1 diabetes mellitus.