Anti-LGI1 LE is considered to have an acute phase that displays the classic limbic encephalitis findings and a prodromal phase, which consists of FBDS with or without sleep and behavior changes [5]. Anti-LGI1 LE has several symptoms that can distinguish it from other forms of autoimmune encephalitis, including hyponatremia, various seizure semiologies (more specifically the faciobrachial dystonic seizures), and various neurocognitive issues [6]. The seizures experienced by patients are found to vary and may range from subclinical to motor to sensory [7]. Here, LGI1 is linked to Hyponatremia.